Oxidative damage causes formation of lipofuscin-like substances in the hippocampus of the senescence-accelerated mouse after kainate treatment

Author:

Kim Hyoung-Chun,Bing Guoying,Jhoo Wang-Kee,Kim Won-Ki,Shin Eun-Joo,Park Eon-Sup,Choi Yong-Soon,Lee Dong-Wook,Shin Chan Young,Ryu Jae Ryun,Ko Kwang Ho

Publisher

Elsevier BV

Subject

Behavioral Neuroscience

Reference68 articles.

1. Perspective of biochemical research in the neuronal ceroid-lipofuscinosis;Alfred-Rider;Am. J. Med. Genet.,1992

2. Morphological evaluation of the lipofuscinolytic effect of acetylhomocysteine thiolactone;Aloj-Totaro;Arch. Gerontol. Geriatr.,1985

3. Extracellular studies on the role of N-methyl-d-aspartate receptors in epileptiform activity recorded from kainic acid lesioned hippocampus;Ashwood;Neurosci. Lett.,1986

4. Hydrogen peroxide mediates amyloid β protein toxicity;Behl;Cell,1994

5. Alzheimer's disease and oxidative stress: implication for novel thrapeutic approaches;Behl;Prog. Neurobiol.,1999

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