Author:
Hidesuke Fukazawa,Pei-Mlng Li,Chii Yamamoto,Yuko Murakami,Satoshi Mizuno,Yoshimasa Uehara
Subject
Pharmacology,Biochemistry
Reference17 articles.
1. Screening of agents which convert “transformed cell morphology” of Rous sarcoma virus-infected rat kidney cells to “normal morphology”: identification of an active agent as herbimycin A and its inhibition of intracellular src kinase;Uehara;Jpn J Cancer Res,1985
2. Phenotyplc change from transformed to normal induced by benzoquinonoid ansamycins accompanies inactivation of p60v-src in rat kidney cells infected with Rous sarcoma virus;Uehara;Mol Cell Biol,1986
3. Inhibition of transforming activity of tyrosine kinase oncogenes by herbimycin A;Uehara;Virology,1988
4. Mechanism of reversion of Rous sarcoma virus transformation by herbimycin A: reduction of total phosphotyrosine levels due to reduced kinase activity and increased turnover of p60v-src;Uehara;Cancer Res,1989
5. Irreversible inhibition of v-src tyrosine kinase activity by herbimycin A and its abrogation by sulfhydryl compounds;Uehara;Biochem Biophys Res Commun,1989
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