The cycad neurotoxic amino acid, ß-N-methylamino-l-alanine (BMAA), elevates intracellular calcium levels in dissociated rat brain cells

Author:

Brownson Delia M,Mabry Tom J,Leslie Steven W

Publisher

Elsevier BV

Subject

Drug Discovery,Pharmacology

Reference56 articles.

1. N-Methylamino-l-alanine in the presence of bicarbonate is an agonist at non-N-methyl-d-aspartate-type receptors;Allen;Journal of Neuroscience,1993

2. The neurotoxin, beta-N-methylamino-l-alanine (BMAA) interacts with the strychnine-insensitive glycine modulatory site of the N-methyl-d-aspartate receptor;Allen;Neuropharmacology,1995

3. Inositol triphosphate and calcium signaling;Berridge;Nature,1993

4. Structure–function studies on N-oxalyl-diamino-dicarboxilic acids and excitatory amino acid receptors: evidence that ß-l-ODAP is a selective non-NMDA agonist;Bridges;Journal of Neuroscience,1989

5. Brownson, D.M., Leslie, S.W., Mabry, T.J., Randall, J.S., 1993. Elevation of intracellular calcium levels in dissociated newborn rat brain cells caused by ß-N-methylamino-l-alanine. In: Proceedings of the 23rd Annual Meeting of the Society of Neuroscience, Abstract 553.13.

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