Involvement of the inhibition of mitochondrial apoptotic, p53, NF-κB pathways and the activation of Nrf2/HO-1 pathway in the protective effects of curcumin against copper sulfate-induced nephrotoxicity in mice-Reference-Cited by-同舟云学术

Involvement of the inhibition of mitochondrial apoptotic, p53, NF-κB pathways and the activation of Nrf2/HO-1 pathway in the protective effects of curcumin against copper sulfate-induced nephrotoxicity in mice

Published:2023-01 Issue: Volume:249 Page:114480
ISSN:0147-6513
Container-title:Ecotoxicology and Environmental Safety
language:en
Short-container-title:Ecotoxicology and Environmental Safety

Author:

Dai Chongshan^ORCID,Li Meng,Liu Yue,Tran Diem Hong,Jiang Haiyang,Tang Shusheng,Shen Jianzhong

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health,Pollution,General Medicine

Reference46 articles.

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3. The molecular mechanisms of copper metabolism and its roles in human diseases;Chen;Pflug. Arch.,2020

4. The mechanisms underlying the cytotoxic effects of copper via differentiated embryonic chondrocyte gene 1;Chen;Int. J. Mol. Sci.,2019

5. Colistin-induced nephrotoxicity in mice involves the mitochondrial, death receptor, and endoplasmic reticulum pathways;Dai;Antimicrob. Agents Chemother.,2014

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