Genistein and tyrphostin AG556 inhibit inwardly-rectifying Kir2.1 channels expressed in HEK 293 cells via protein tyrosine kinase inhibition-Reference-Cited by-同舟云学术

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Genistein and tyrphostin AG556 inhibit inwardly-rectifying Kir2.1 channels expressed in HEK 293 cells via protein tyrosine kinase inhibition

Published:2011-08 Issue:8 Volume:1808 Page:1993-1999
ISSN:0005-2736
Container-title:Biochimica et Biophysica Acta (BBA) - Biomembranes
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Biomembranes

Author:

Zhang De-Yong,Wu Wei,Deng Xiu-Ling,Lau Chu-Pak,Li Gui-Rong

Funder

Research Grant Council of Hong Kong

Sun Chieh Yeh Heart Foundation of Hong Kong

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry,Biophysics

Reference43 articles.

1. Inward rectifiers in the heart: an update on IK1;Lopatin;J. Mol. Cell. Cardiol.,2001

2. Merging functional studies with structures of inward-rectifier K(+) channels;Bichet;Nat. Rev. Neurosci.,2003

3. Primary structure and functional expression of a mouse inward rectifier potassium channel;Kubo;Nature,1993

4. Channelopathies: Kir2.1 mutations jeopardize many cell functions;Jongsma;Curr. Biol.,2001

5. The inward rectifier current (IK1) controls cardiac excitability and is involved in arrhythmogenesis;Dhamoon;Heart Rhythm,2005

Cited by 15 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Proteomic Analysis of the Functional Inward Rectifier Potassium Channel (Kir) 2.1 Reveals Several Novel Phosphorylation Sites;Biochemistry;2021-10-22

2. Inhibitory Effects of Genistein on Vascular Smooth Muscle Cell Proliferation Induced by Ox-LDL: Role of BKCa Channels;Analytical Cellular Pathology;2020-12-13

3. The beneficial health effects of flavonoids on the cardiovascular system: Focus on K+ channels;Pharmacological Research;2020-02

4. Regulation of Kir2.1 Function Under Shear Stress and Cholesterol Loading;Journal of the American Heart Association;2018-03-06

5. Effects of equol on multiple K+ channels stably expressed in HEK 293 cells;PLOS ONE;2017-08-23

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