Down regulated connexin26 at different postnatal stage displayed different types of cellular degeneration and formation of organ of Corti
Author:
Funder
National Nature Science Foundation of China
973 Program
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference20 articles.
1. Connexin 26 mutations in hereditary non-syndromic sensorineural deafness;Kelsell;Nature,1997
2. Contribution of connexin26 (GJB2) mutations and founder effect to non-syndromic hearing loss in India;RamShankar;J. Med. Genet.,2003
3. Mutations in the connexin 26 gene (GJB2) among Ashkenazi Jews with nonsyndromic recessive deafness;Morell;N. Engl. J. Med.,1998
4. Gap-junction channels dysfunction in deafness and hearing loss;Martinez;Antioxid. Redox Signal.,2009
5. Targeted ablation of connexin26 in the inner ear epithelial gap junction network causes hearing impairment and cell death;Cohen-Salmon;Curr. Biol.,2002
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