Cardiac-specific deletion of heat shock protein 60 induces mitochondrial stress and disrupts heart development in mice-Reference-Cited by-同舟云学术

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Cardiac-specific deletion of heat shock protein 60 induces mitochondrial stress and disrupts heart development in mice

Published:2024-05 Issue: Volume:710 Page:149883
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Shen Tao,Wang Shuting,Huang Can,Zhu Siting^ORCID,Zhu Xiangbin^ORCID,Li Na,Wang Hong,Huang Lei,Ren Mingming,Han Zhen,Ge Jianjun,Chen Ze'e,Ouyang Kunfu^ORCID

Funder

China Postdoctoral Science Foundation

CAS Shenzhen-Hong Kong Institute of Brain Science

Department of Science and Technology of Guangdong Province

Shenzhen Science and Technology Innovation Committee

National Natural Science Foundation of China

Ministry of Science and Technology of the People's Republic of China

Shenzhen Basic Research Program

Publisher

Elsevier BV

Reference62 articles.

1. Basics of cardiac development for the understanding of congenital heart malformations;Gittenberger-de Groot;Pediatr. Res.,2005

2. Congenital heart disease in pediatric patients: recognizing the undiagnosed and managing complications in the emergency department;Judge;Pediatr. Emerg. Med. Pract.,2016

3. Inactivation of the hereditary spastic paraplegia-associated Hspd1 gene encoding the Hsp60 chaperone results in early embryonic lethality in mice;Christensen;Cell Stress Chaperones,2010

4. Heat shock protein 60 regulates yolk sac erythropoiesis in mice;Duan;Cell Death Dis.,2019

5. Mitochondrial fusion directs cardiomyocyte differentiation via calcineurin and Notch signaling;Kasahara;Science,2013

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