MicroRNA-27a reduces mutant hutingtin aggregation in an in vitro model of Huntington's disease-Reference-Cited by-同舟云学术

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MicroRNA-27a reduces mutant hutingtin aggregation in an in vitro model of Huntington's disease

Published:2017-06 Issue:2 Volume:488 Page:316-321
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Ban Jae-Jun,Chung Jin-Young,Lee Mijung,Im Wooseok,Kim Manho

Funder

National Research Foundation of Korea (NRF)

Ministry of Science, ICT & Future Planning

SNUH

National Research Foundation of Korea

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference42 articles.

1. Huntingtin and the molecular pathogenesis of Huntington's disease. Fourth in molecular medicine review series;Landles;EMBO Rep.,2004

2. Trinucleotide repeat length instability and age of onset in Huntington's disease;Duyao;Nat. Genet.,1993

3. Correlation between the onset age of Huntington's disease and length of the trinucleotide repeat in IT-15;Stine;Hum. Mol. Genet.,1993

4. Regional and cellular gene expression changes in human Huntington's disease brain;Hodges;Hum. Mol. Genet.,2006

5. MicroRNAs: genomics, biogenesis, mechanism, and function;Bartel;Cell,2004

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