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Excessive N-acetylcysteine exaggerates glutathione redox homeostasis and apoptosis during acetaminophen exposure in Huh-7 human hepatoma cells

  • Published:2023-10 Issue: Volume:676 Page:66-72
  • ISSN:0006-291X
  • Container-title:Biochemical and Biophysical Research Communications
  • language:en
  • Short-container-title:Biochemical and Biophysical Research Communications

Author:

Aki Toshihiko,Tanaka Hiroki,Funakoshi Takeshi,Unuma Kana,Uemura Koichi

Funder

Japan Society for the Promotion of Science

Ministry of Education, Culture, Sports, Science and Technology

Tokyo Medical and Dental University

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference28 articles.

1. Acetaminophen hepatotoxicity;Ramachandran;Semin. Liver Dis.,2019

2. Emerging and established modes of cell death during acetaminophen-induced liver injury;Jaeschke;Arch. Toxicol.,2019

3. Acetaminophen-induced apoptosis: facts versus fiction;Jaeschke;J. Clin. Transl. Res.,2020

4. The role of apoptosis in acetaminophen hepatotoxicity;Jaeschke;Food Chem. Toxicol.,2018

5. Comparative analysis of phase I and II enzyme activities in 5 hepatic cell lines identifies Huh-7 and HCC-T cells with the highest potential to study drug metabolism;Lin;Arch. Toxicol.,2012

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