Angiotensin II increases the cholesterol content of foam cells via down-regulating the expression of ATP-binding cassette transporter A1
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference18 articles.
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4. Decreased cellular cholesterol efflux is a common cause of familial hypoalphalipoproteinemia: role of the ABCA1 gene mutations;Mott;Atherosclerosis,2000
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1. Angiotensin II induces the expression of periostin to promote foam cell formation in oxLDL-treated macrophages;International Journal of Cardiology;2022-01
2. α-Hederin Induces Apoptosis, Membrane Permeabilization and Morphologic Changes in Two Cancer Cell Lines Through a Cholesterol-Dependent Mechanism;Planta Medica;2016-08-30
3. Angiotensin-(1-7) upregulates expression of adenosine triphosphate-binding cassette transporter A1 and adenosine triphosphate-binding cassette transporter G1 through the Mas receptor through the liver X receptor alpha signalling pathway in THP-1 macrophag;Clinical and Experimental Pharmacology and Physiology;2014-12
4. Excess nitric oxide impairs liver X receptor α-ATP-binding cassette transporter A1-dependent cholesterol efflux in macrophage foam cells;Journal of Cellular Physiology;2013-07
5. Valsartan and Telmisartan Abrogate Angiotensin II–induced Downregulation of ABCA1 Expression Via AT1 Receptor, Rather than AT2 Receptor or PPARγ Activation;Journal of Cardiovascular Pharmacology;2012-06
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