In the rat, tumor necrosis factor α administration results in an increase in both UCP2 and UCP3 mRNAs in skeletal muscle: a possible mechanism for cytokine-induced thermogenesis?-Reference-Cited by-同舟云学术

In the rat, tumor necrosis factor α administration results in an increase in both UCP2 and UCP3 mRNAs in skeletal muscle: a possible mechanism for cytokine-induced thermogenesis?

Published:1998-12-04 Issue:3 Volume:440 Page:348-350
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Busquets Sı́lvia,Sanchı́s Daniel,Alvarez Belén,Ricquier Daniel,López-Soriano Francisco J,Argilés Josep M

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(98)01485-9/fullpdf

Reference19 articles.

1. Uncoupling protein-2: a novel gene linked to obesity and hyperinsulinemia

2. Cloning and Characterization of an Uncoupling Protein Homolog: A Potential Molecular Mediator of Human Thermogenesis

3. Uncoupling Protein-3 Is a Mediator of Thermogenesis Regulated by Thyroid Hormone, β3-Adrenergic Agonists, and Leptin

4. UCP3: An Uncoupling Protein Homologue Expressed Preferentially and Abundantly in Skeletal Muscle and Brown Adipose Tissue

5. The Human Uncoupling Protein-3 Gene

Cited by 90 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. TNF-α-1031T/C gene polymorphism as a predictor of malnutrition in patients with gastric cancer;Frontiers in Nutrition;2023-07-18

2. Regulation of mitochondrial temperature in health and disease;Pflügers Archiv - European Journal of Physiology;2022-07-02

3. Cancer cachexia and its pathophysiology: links with sarcopenia, anorexia and asthenia;Journal of Cachexia, Sarcopenia and Muscle;2020-03-06

4. Modulation of Oxidative Stress in Heart Disease by Uncoupling Proteins;Modulation of Oxidative Stress in Heart Disease;2019

5. The tumor secretory factor ZAG promotes white adipose tissue browning and energy wasting;The FASEB Journal;2018-03-23