Early expression of the BM88 antigen during neuronal differentiation of P19 embryonal carcinoma cells
Author:
Affiliation:
1. Department of BiochemistryHellenic Pasteur Institute127 Vassilissis Sofias Avenue115 21AthensGreece
Publisher
Wiley
Subject
Developmental Biology,Developmental Neuroscience
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1016/S0736-5748%2899%2900101-X
Reference44 articles.
1. Activation of the gene for Atrial Natriuretic Factor during in vitro cardiac myogenesis by P19 ebryonal carcinoma cells;Boer H.P.;Exp. Cell Res.,1993
2. Differential expression of gap junctions in neurons and astrocytes derived from P19 embryonal carcinoma cells
3. Overexpression of Stra13, a novel retinoic acid-inducible gene of the basic helix–loop–helix family, inhibits mesodermal and promotes neuronal differentiation of P19 cells
4. Inhibition of MAP2 expression affects both morphological and cell division phenotypes of neuronal differentiation
5. Spatial regulation of axonal glycoprotein expression on subsets of embryonic spinal neurons
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1. Cend1, a Story with Many Tales: From Regulation of Cell Cycle Progression/Exit of Neural Stem Cells to Brain Structure and Function;Stem Cells International;2019-05-02
2. A method for generating high-yield enriched neuronal cultures from P19 embryonal carcinoma cells;Journal of Neuroscience Methods;2012-02
3. Characterization of the BM88 promoter and identification of an 88 bp fragment sufficient to drive neurone-specific expression;Journal of Neurochemistry;2005-07-20
4. BM88 is an early marker of proliferating precursor cells that will differentiate into the neuronal lineage;European Journal of Neuroscience;2004-11
5. Failure to express GAP-43 leads to disruption of a multipotent precursor and inhibits astrocyte differentiation;Molecular and Cellular Neuroscience;2004-07
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