The DNA double-strand break response pathway: becoming more BRCAish than ever-Reference-Cited by-同舟云学术

The DNA double-strand break response pathway: becoming more BRCAish than ever

Published:2004-08 Issue:8-9 Volume:3 Page:935-944
ISSN:1568-7864
Container-title:DNA Repair
language:en
Short-container-title:DNA Repair

Author:

Ting Nicholas S.Y.,Lee Wen-Hwa

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference92 articles.

1. A strong candidate for the breast and ovarian cancer susceptibility gene BRCA1;Miki;Science,1994

2. BRCA1-associated tumorigenesis: what have we learned from knockout mice?;Brodie;Trends Genet.,2001

3. Roles of BRCA1 in DNA damage repair: a link between development and cancer;Deng;Hum. Mol. Genet.,2003

4. Homology-directed DNA repair, mitomycin C resistance, and chromosome stability is restored with correction of a Brca1 mutation;Moynahan;Cancer Res.,2001

5. Lessons learned from BRCA1 and BRCA2;Zheng;Oncogene,2000

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