NMDA receptor antagonism modifies the synergistic regulation of striatal tachykinin gene expression induced by dopamine D1 and serotonin2 receptor stimulation following neonatal dopamine depletion-Reference-Cited by-同舟云学术

NMDA receptor antagonism modifies the synergistic regulation of striatal tachykinin gene expression induced by dopamine D1 and serotonin2 receptor stimulation following neonatal dopamine depletion

Published:2001-09 Issue:1 Volume:93 Page:90-94
ISSN:0169-328X
Container-title:Molecular Brain Research
language:en
Short-container-title:Molecular Brain Research

Author:

Campbell B.M.,Walker P.D.

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Molecular Biology

Reference29 articles.

1. Stimulated serotonin release from hyperinnervated terminals subsequent to neonatal dopamine depletion regulates striatal tachykinin, but not enkephalin gene expression;Basura;Mol. Brain Res.,2000

2. Effects of dizocilpine (MK801) on noradrenaline, serotonin, and dopamine release and reuptake;Callado;Neuroreport,2000

3. MK801 blocks dopamine D1- but not serotonin 2A-receptor stimulation of PPT mRNA expression in the rat striatum in organotypic slice cultures;Campbell;Neuroreport,2001

4. B.M. Campbell, P.J. Gresch, P.D. Walker, Neonatal dopamine depletion reveals a synergistic mechanism of mRNA regulation that is mediated by dopamineD1 and serotonin2 receptors and is targeted to tachykinin neurons of the dorsomedial striatum. Neuroscience (2001) (in press).

5. Effects of MK-801 on D1 dopamine receptor-mediated immediate early gene expression in the dopamine-depleted striatum;Ganguly;Brain Res.,2000

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