Post-stroke administration of H2 relaxin reduces functional deficits, neuronal apoptosis and immune cell infiltration into the mouse brain-Reference-Cited by-同舟云学术

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Post-stroke administration of H2 relaxin reduces functional deficits, neuronal apoptosis and immune cell infiltration into the mouse brain

Published:2023-01 Issue: Volume:187 Page:106611
ISSN:1043-6618
Container-title:Pharmacological Research
language:en
Short-container-title:Pharmacological Research

Author:

Truong Shirley H.T.,Bonnici Benjamin,Rupasinghe Samoda,Kemp-Harper Barbara K.,Samuel Chrishan S.,Broughton Brad R.S.

Funder

Monash University

Monash Biomedicine Discovery Institute, Monash University

Publisher

Elsevier BV

Subject

Pharmacology

Reference56 articles.

1. Global, regional, and national burden of stroke and its risk factors, 1990–2019: a systematic analysis for the Global Burden of Disease Study 2019;Feigin;Lancet Neurol.,2021

2. A. Majid, Neuroprotection in stroke: past, present, and future, ISRN Neurol 2014 (2014) 515716.

3. The science of stroke: mechanisms in search of treatments;Moskowitz;Neuron,2010

4. Tissue plasminogen activator for acute ischemic stroke;N. Engl. J. Med.,1995

5. The role of stroke nurses in thrombolysis administration in Australia and the United Kingdom: a cross-sectional survey of current practice;Hamilton;J. Clin. Nurs.,2022

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2. CD300LF⁺ microglia impede the neuroinflammation following traumatic brain injury by inhibiting STING pathway;CNS Neuroscience & Therapeutics;2024-07

3. INSL3 suppresses LPS-induced inflammation in N9 microglia cells;Cukurova Medical Journal;2024-06-30

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