Ascorbate attenuates trimethyltin-induced oxidative burden and neuronal degeneration in the rat hippocampus by maintaining glutathione homeostasis

Author:

Shin E.-J.,Suh S.K.,Lim Y.K.,Jhoo W.-K.,Hjelle O.P.,Ottersen O.P.,Shin C.Y.,Ko K.H.,Kim W.-K.,Kim D.S.,Chun W.,Ali S.,Kim H.-C.

Publisher

Elsevier BV

Subject

General Neuroscience

Reference59 articles.

1. Reactive oxygen species formation as a biomarker of methylmercury and trimethyltin neurotoxicity;Ali;Neurotoxicology,1992

2. Trimethyltin-induced loss of NMDA and kainate receptors in the rat brain;Andersson;Amino Acids,1995

3. Neurodegenerative disorders in humans;Bains;Brain Res Rev,1997

4. The effects of various levels of ascorbic acid on the response of the ODS rat to trimethyltin;Bannon;Neurotoxicology,1993

5. The behavioral and neuropathologic sequelae of intoxication by trimethyltin compounds in the rat;Brown;Am J Pathol,1979

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