Enhanced nonsynaptic epileptiform activity in the dentate gyrus after kainate-induced status epilepticus

Author:

Nogueira G.S.,Santos L.E.C.,Rodrigues A.M.,Scorza C.A.,Scorza F.A.,Cavalheiro E.A.,de Almeida A.-C.G.

Funder

FAPEMIG

FAPESP

CNPq

PROCAD/CAPES

CAPES

INCT of Translational Neuroscience

Publisher

Elsevier BV

Subject

General Neuroscience

Reference28 articles.

1. Mechanistic hypotheses for nonsynaptic epileptiform activity induction and its transition from the interictal to ictal state–computational simulation;Almeida;Epilepsia,2008

2. Biophysical aspects of the nonsynaptic epileptiform activity;Almeida,2011

3. Status epilepticus induces a particular microglial activation state characterized by enhanced purinergic signaling;Avignone;J Neurosci,2008

4. Ben-Ari Y (2012) Kainate and temporal lobe epilepsies: 3 decades of progress. In: Jasper’s basic mechanisms of the epilepsies, 4th edition (Noebel Jl, Avoli M, Rogawski MA, Olsen RW, Delgado-Escueta AV, ed). pp 432–453 Bethesda (MD): National Center for Biotechnology Information (US). Chapter 33.

5. Epileptogenic action of intra-amygdaloid injection of kainic acid;Ben-Ari;C R Acad Sci Hebd Seances Acad Sci D,1978

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