Neprilysin activity is increased in metabolic dysfunction-associated steatotic liver disease and normalizes after bariatric surgery or GLP-1 therapy-Reference-Cited by-同舟云学术

Neprilysin activity is increased in metabolic dysfunction-associated steatotic liver disease and normalizes after bariatric surgery or GLP-1 therapy

Published:2023-11 Issue:11 Volume:26 Page:108190
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Kjeldsen Sasha A.S.,Gluud Lise L.,Werge Mikkel P.,Pedersen Julie S.,Bendtsen Flemming,Alexiadou Kleopatra,Tan Tricia,Torekov Signe S.,Iepsen Eva W.,Jensen Nicole J.,Richter Michael M.,Goetze Jens P.,Rungby Jørgen,Hartmann Bolette,Holst Jens J.,Holst Birgitte,Holt Joachim,Gustafsson Finn,Madsbad Sten,Svane Maria S.,Bojsen-Møller Kirstine N.,Wewer Albrechtsen Nicolai J.

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference48 articles.

1. The purification and specificity of a neutral endopeptidase from rabbit kidney brush border;Kerr;Biochem. J.,1974

2. Neprilysin expression and functions in development, ageing and disease;Nalivaeva;Mech. Ageing Dev.,2020

3. Sacubitril/valsartan augments postprandial plasma concentrations of active GLP-1 when combined with sitagliptin in men;Wewer Albrechtsen;J. Clin. Endocrinol. Metab.,2019

4. Acute effects on glucose tolerance by neprilysin inhibition in patients with type 2 diabetes;Wewer Albrechtsen;Diabetes Obes. Metab.,2022

5. Dual angiotensin receptor and neprilysin inhibition as an alternative to angiotensin-converting enzyme inhibition in patients with chronic systolic heart failure: rationale for and design of the Prospective comparison of ARNI with ACEI to Determine Impact

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Hepatic Amyloid Beta-42-Metabolizing Proteins in Liver Steatosis and Metabolic Dysfunction-Associated Steatohepatitis;International Journal of Molecular Sciences;2024-08-12

2. Gut-specific Neprilysin Deletion Protects Against Fat-induced Insulin Secretory Dysfunction in Male Mice;Endocrinology;2024-07-01