Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome-Reference-Cited by-同舟云学术

Evidence that increased Kcnj6 gene dose is necessary for deficits in behavior and dentate gyrus synaptic plasticity in the Ts65Dn mouse model of Down syndrome

Published:2017-07 Issue: Volume:103 Page:1-10
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Kleschevnikov Alexander M.,Yu Jessica,Kim Jeesun,Lysenko Larisa V.,Zeng Zheng,Yu Y. Eugene,Mobley William C.

Funder

Jerome Lejeune Foundation

NIH

Publisher

Elsevier BV

Subject

Neurology

Reference104 articles.

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4. The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome;Belichenko;J. Neurosci.,2009

5. Down syndrome cognitive phenotypes modeled in mice trisomic for all HSA 21 homologues;Belichenko;PLoS One,2015

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