Friedreich ataxia- pathogenesis and implications for therapies-Reference-Cited by-同舟云学术

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Friedreich ataxia- pathogenesis and implications for therapies

Published:2019-12 Issue: Volume:132 Page:104606
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Delatycki Martin B.^ORCID,Bidichandani Sanjay I.

Publisher

Elsevier BV

Subject

Neurology

Reference141 articles.

1. Mitochondrial energy imbalance and lipid peroxidation cause cell death in Friedreich's ataxia;Abeti;Cell Death Dis.,2016

2. Drug repositioning screening identifies etravirine as a potential therapeutic for friedreich’s ataxia;Alfedi;Mov. Disord.,2019

3. GAA repeat expansion mutation mouse models of Friedreich ataxia exhibit oxidative stress leading to progressive neuronal and cardiac pathology;Al-Mahdawi;Genomics.,2006

4. Iron in Friedreich Ataxia: a central role in the pathophysiology or an epiphenomenon?;Alsina;Pharmaceuticals (Basel),2018

5. A novel GAA-repeat-expansion-based mouse model of Friedreich’s ataxia;Anjomani Virmouni;Dis. Model. Mech.,2015

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1. A multiple animal and cellular models approach to study frataxin deficiency in Friedreich Ataxia;Biochimica et Biophysica Acta (BBA) - Molecular Cell Research;2024-10

2. Unusual Age‐Dependent Behavior of Leukocytes Telomere Length in Friedreich's Ataxia;Movement Disorders;2024-09-05

3. Generation of genetically modified Friedreich’s ataxia induced pluripotent stem cell lines and isogenic control lines carrying an inducible neurogenin-2 expression cassette;Stem Cell Research;2024-09

4. New and Emerging Drug and Gene Therapies for Friedreich Ataxia;CNS Drugs;2024-08-08

5. Emerging therapies in hereditary ataxias;Trends in Molecular Medicine;2024-08

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