Early deficits in GABA inhibition parallels an increase in L-type Ca2+ currents in the jaw motor neurons of SOD1G93A mouse model for ALS-Reference-Cited by-同舟云学术

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Early deficits in GABA inhibition parallels an increase in L-type Ca2+ currents in the jaw motor neurons of SOD1G93A mouse model for ALS

Published:2023-02 Issue: Volume:177 Page:105992
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Venugopal Sharmila,Ghulam-Jelani Zohal,Ahn In Sook,Yang Xia,Wiedau Martina,Simmons Dwayne,Chandler Scott H.

Funder

University of California, Los Angeles

National Institute on Deafness and Other Communication Disorders

National Institute of Neurological Disorders and Stroke

Publisher

Elsevier BV

Subject

Neurology

Reference107 articles.

1. Astrocyte regulation of synaptic behavior;Allen;Annu. Rev. Cell Dev. Biol.,2014

2. Synaptic plasticity: astrocytes wrap it up;Allen;Curr. Biol.,2014

3. Locomotor deficits in a mouse model of ALS are paralleled by loss of V1-interneuron connections onto fast motor neurons;Allodi;Nat. Commun.,2021

4. Early signs of motoneuron vulnerability in a disease model system: characterization of transverse slice cultures of spinal cord isolated from embryonic ALS mice;Avossa;Neuroscience,2006

5. Synaptic restoration by cAMP/PKA drives activity-dependent neuroprotection to motoneurons in ALS;Baczyk;J. Exp. Med.,2020

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