Substitutions in the Escherichia coli RNA polymerase inhibitor T7 Gp2 that allow inhibition of transcription when the primary interaction interface between Gp2 and RNA polymerase becomes compromised-Reference-Cited by-同舟云学术

Substitutions in the Escherichia coli RNA polymerase inhibitor T7 Gp2 that allow inhibition of transcription when the primary interaction interface between Gp2 and RNA polymerase becomes compromised

Published:2012-11-01 Issue:11 Volume:158 Page:2753-2764
ISSN:1350-0872
Container-title:Microbiology
language:en
Short-container-title:

Author:

Shadrin Andrey¹,Sheppard Carol¹,Severinov Konstantin²³⁴,Matthews Steve¹,Wigneshweraraj Sivaramesh¹

Affiliation:

1. Section of Microbiology and MRC Centre for Molecular Bacteriology and Infection, Imperial College London, London SW7 2AZ, UK

2. Institute of Gene Biology, Russian Academy of Sciences, Moscow 119334, Russia

3. Institute of Molecular Genetics, Russian Academy of Sciences, Moscow 123182, Russia

4. Waksman Institute for Microbiology and Department of Molecular Biology and Biochemistry, Rutgers, The State University of New Jersey, Piscataway, NJ 08854, USA

Publisher

Microbiology Society

Subject

Microbiology

Reference24 articles.

1. The SWISS-MODEL workspace: a web-based environment for protein structure homology modelling

2. Structural Basis for Converting a General Transcription Factor into an Operon-Specific Virulence Regulator

3. The regulation of bacterial transcription initiation

4. Marker rescue and partial replication of bacteriophage T7 DNA.

5. T7 phage protein Gp2 inhibits the Escherichia coli RNA polymerase by antagonizing stable DNA strand separation near the transcription start site

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