Transthyretin and Receptor for Advanced Glycation End Product’s Differential Levels Associated with the Pathogenesis of Rheumatoid Arthritis-Reference-Cited by-同舟云学术

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Transthyretin and Receptor for Advanced Glycation End Product’s Differential Levels Associated with the Pathogenesis of Rheumatoid Arthritis

Published:2021-10 Issue: Volume:Volume 14 Page:5581-5596
ISSN:1178-7031
Container-title:Journal of Inflammation Research
language:en
Short-container-title:JIR

Author:

Monu ,Agnihotri Prachi,Saquib Mohd,Sarkar Ashish,Chakraborty Debolina,Kumar Uma^ORCID,Biswas Sagarika^ORCID

Publisher

Informa UK Limited

Subject

Immunology,Immunology and Allergy

Reference43 articles.

1. Rheumatoid arthritis

2. WHO. Rheumatoid arthritis; 2018. Available from: www.who.int/chp/topics/rheumatic/en/. Accessed October 8, 2021.

3. Rheumatoid arthritis: pathological mechanisms and modern pharmacologic therapies

4. The Clinical Significance of Posttranslational Modification of Autoantigens

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The Potential Influence of Advanced Glycation End Products and (s)RAGE in Rheumatic Diseases;International Journal of Molecular Sciences;2023-02-02

2. Upregulation of leucine-rich alpha-2 glycoprotein: A key regulator of inflammation and joint fibrosis in patients with severe knee osteoarthritis;Frontiers in Immunology;2022-12-07

3. Pathophysiology of RAGE in inflammatory diseases;Frontiers in Immunology;2022-07-29

4. Blood transthyretin for predicting immunoglobulin A vasculitis nephritis outcome in children;International Immunopharmacology;2022-07

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