N- acetylcysteine attenuates the development of cardiac fibrosis and remodeling in a mouse model of heart failure-Reference-Cited by-同舟云学术

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N- acetylcysteine attenuates the development of cardiac fibrosis and remodeling in a mouse model of heart failure

Published:2016-04 Issue:7 Volume:4 Page:e12757
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiol Rep

Author:

Giam Beverly¹²,Chu Po-Yin¹,Kuruppu Sanjaya³,Smith A. Ian³,Horlock Duncan¹,Kiriazis Helen¹,Du Xiao-Jun¹,Kaye David M.¹⁴,Rajapakse Niwanthi W.¹⁵

Affiliation:

1. Baker IDI Heart and Diabetes Institute; Melbourne Australia

2. Central Clinical School; Monash University; Melbourne Australia

3. Department of Biochemistry; Monash University; Melbourne Australia

4. Department of Medicine; Monash University; Melbourne Australia

5. Department of Physiology; Monash University; Melbourne Australia

Funder

Baker IDI Heart and Diabetes Institute

National Health and Medical Research Council

State Government of Victoria

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.12757/fullpdf

Reference34 articles.

1. Neutral sphingomyelinase inhibition participates to the benefits of N-acetylcysteine treatment in post-myocardial infarction failing heart rats;Adamy;J. Mol. Cell. Cardiol.,2007

2. Oxidative stress triggers cardiac fibrosis in the heart of diabetic rats;Aragno;Endocrinology,2008

3. N-acetylcysteine treatment normalizes serum tumor necrosis factor-alpha level and hinders the progression of cardiac injury in hypertensive rats;Bourraindeloup;Circulation,2004

4. Oxidative stress-mediated cardiac cell death is a major determinant of ventricular dysfunction and failure in dog dilated cardiomyopathy;Cesselli;Circ. Res.,2001

5. Bone marrow-derived cells contribute to fibrosis in the chronically failing heart;Chu;Am. J. Pathol.,2010

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