CLIC1 null mice demonstrate a role for CLIC1 in macrophage superoxide production and tissue injury-Reference-Cited by-同舟云学术

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CLIC1 null mice demonstrate a role for CLIC1 in macrophage superoxide production and tissue injury

Published:2017-03 Issue:5 Volume:5 Page:e13169
ISSN:2051-817X
Container-title:Physiological Reports
language:en
Short-container-title:Physiol Rep

Author:

Ulmasov Barbara¹,Bruno Jonathan¹²,Oshima Kiyoko³,Cheng Yao-Wen²,Holly Stephen P.⁴,Parise Leslie V.⁴,Egan Terrance M.⁵,Edwards John C.¹²⁵

Affiliation:

1. Department of Internal Medicine; Saint Louis University; St. Louis Missouri

2. UNC Kidney Center; University of North Carolina; Chapel Hill North Carolina

3. Department of Pathology; Medical College of Wisconsin; Milwaukee Wisconsin

4. Department of Biochemistry and Biophysics; University of North Carolina; Chapel Hill North Carolina

5. Department of Pharmacology and Physiology; Saint Louis University; St. Louis Missouri

Funder

National Institutes of Health

Publisher

Wiley

Subject

Physiology (medical),Physiology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.13169/fullpdf

Reference60 articles.

1. The role of an astrocytic NADPH oxidase in the neurotoxicity of amyloid beta peptides;Abramov;Philos. Trans. R. Soc. Lond. B Biol. Sci.,2005

2. Members of the chloride intracellular ion channel protein family demonstrate glutaredoxin-like enzymatic activity;Al Khamici;PLoS ONE,2015

3. Chloride intracellular channel 1 (CLIC1): Sensor and effector during oxidative stress;Averaimo;FEBS Lett.,2010

4. elegans CLIC-like protein required for intracellular tube formation and maintenance;Berry;Science,2003

5. Identification of a novel member of the chloride intracellular channel gene family (CLIC5) that associates with the actin cytoskeleton of placental microvilli;Berryman;Mol. Biol. Cell,2000

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