Trypanosoma Cruzi Sensitizes Mice to Fulminant Seb-Induced Shock: Overrelease of Inflammatory Cytokines and Independence of Chagas' Disease or TCR V??-Usage-Reference-Cited by-同舟云学术

Trypanosoma Cruzi Sensitizes Mice to Fulminant Seb-Induced Shock: Overrelease of Inflammatory Cytokines and Independence of Chagas' Disease or TCR V??-Usage

Published:2003-02 Issue:2 Volume:19 Page:163-168
ISSN:1073-2322
Container-title:Shock
language:en
Short-container-title:Shock

Author:

Paiva Cl??udia N.,Pyrrho Alexandre S.,Lannes-Vieira Joseli,Vacchio Melanie,Soares Milena B. P.,Gattass Cerli R.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference35 articles.

1. Immunoregulatory mechanisms of T-cell-dependent shock induced by a bacterial superantigen in mice

2. Superantigen and endotoxin synergize in the induction of lethal shock

3. Glucocorticoid-mediated control of the activation and clonal deletion of peripheral T cells in vivo.

4. T cell control of staphylococcal enterotoxin B (SEB) lethal sensitivity in mice: CD4+ CD45RBbright / CD4+ CD45RBdim balance defines susceptibility to SEB toxicity

5. T cell-mediated lethal shock triggered in mice by the superantigen staphylococcal enterotoxin B: critical role of tumor necrosis factor.

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