Sevoflurane Postconditioning Reduces Apoptosis by Activating the JAK-STAT Pathway After Transient Global Cerebral Ischemia in Rats-Reference-Cited by-同舟云学术

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Sevoflurane Postconditioning Reduces Apoptosis by Activating the JAK-STAT Pathway After Transient Global Cerebral Ischemia in Rats

Published:2017-01 Issue:1 Volume:29 Page:37-45
ISSN:0898-4921
Container-title:Journal of Neurosurgical Anesthesiology
language:en
Short-container-title:

Author:

Kim Hyun-Chang,Kim Eugene,Bae Jung Il,Lee Kook Hyun,Jeon Young-Tae,Hwang Jung-Won,Lim Young-Jin,Min Seong-Won,Park Hee-Pyoung

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Anesthesiology and Pain Medicine,Neurology (clinical),Surgery

Reference50 articles.

1. Xenon and sevoflurane protect against brain injury in a neonatal asphyxia model;Luo;Anesthesiology,2008

2. A combination of sevoflurane postconditioning and albumin increases Bcl-2 expression after transient global cerebral ischemia compared with either sevoflurane postconditioning or albumin alone;Jeon;J Neurosurg Anesthesiol,2013

3. Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury via PI3K/Akt pathway;Wang;Brain Res,2010

4. Sevoflurane post-conditioning increases nuclear factor erythroid 2-related factor and haemoxygenase-1 expression via protein kinase C pathway in a rat model of transient global cerebral ischaemia;Lee;Br J Anaesth,2015

5. The Jak-STAT pathway;Imada;Mol Immunol,2000

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