Aggregate distribution in frontal and motor cortex in Huntington??s disease brain
Author:
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
General Neuroscience
Reference24 articles.
1. Trinucleotide Repeats: Mechanisms and Pathophysiology
2. Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin [published erratum appears in Hum Mol Genet 1999 May;8(5):943]
3. Huntington's Disease Intranuclear Inclusions Contain Truncated, Ubiquitinated Huntingtin Protein
4. Huntingtin aggregation and toxicity in Huntington's disease
5. Tissue-Specific Proteolysis of Huntingtin (htt) in Human Brain: Evidence of Enhanced Levels of N- and C-Terminal htt Fragments in Huntington's Disease Striatum
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2. N-terminal mutant huntingtin deposition correlates with CAG repeat length and symptom onset, but not neuronal loss in Huntington's disease;Neurobiology of Disease;2022-11
3. N-terminal mutant Huntingtin deposition correlates with CAG repeat length and disease onset, but not neuronal loss in Huntington’s disease;2022-05-04
4. The distribution and density of Huntingtin inclusions across the Huntington disease neocortex: regional correlations with Huntingtin repeat expansion independent of pathologic grade;Acta Neuropathologica Communications;2022-04-19
5. Epigenetic dysregulation of hairy and enhancer of split 4 (HES4) is associated with striatal degeneration in postmortem Huntington brains;Human Molecular Genetics;2014-12-05
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