M2A and M2C Macrophage Subsets Ameliorate Inflammation and Fibroproliferation in Acute Lung Injury Through Interleukin 10 Pathway-Reference-Cited by-同舟云学术

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M2A and M2C Macrophage Subsets Ameliorate Inflammation and Fibroproliferation in Acute Lung Injury Through Interleukin 10 Pathway

Published:2017-07 Issue:1 Volume:48 Page:119-129
ISSN:1073-2322
Container-title:Shock
language:en
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Author:

Tang Lunxian,Zhang Hua,Wang Chunmei,Li Hongqiang,Zhang Qian,Bai Jianwen

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Critical Care and Intensive Care Medicine,Emergency Medicine

Reference26 articles.

1. Acute respiratory distress syndrome: the berlin definition;Ranieri;JAMA,2012

2. The berlin definition of ards: an expanded rationale, justification, and supplementary material;Ferguson;Intensive Care Med,2012

3. The acute respiratory distress syndrome: pathogenesis and treatment;Matthay;Annu Rev Pathol,2011

4. Cd4+cd25+foxp3+ tregs resolve experimental lung injury in mice and are present in humans with acute lung injury;D’Alessio;J Clin Invest,2009

5. Regulatory t cells reduce acute lung injury fibroproliferation by decreasing fibrocyte recruitment;Garibaldi;Am J Respir Cell and Mol Biol,2013

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