A member of the gut mycobiota modulates host purine metabolism exacerbating colitis in mice-Reference-Cited by-同舟云学术

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A member of the gut mycobiota modulates host purine metabolism exacerbating colitis in mice

Published:2017-03-08 Issue:380 Volume:9 Page:
ISSN:1946-6234
Container-title:Science Translational Medicine
language:en
Short-container-title:Sci. Transl. Med.

Author:

Chiaro Tyson R.¹^ORCID,Soto Ray¹,Zac Stephens W.¹^ORCID,Kubinak Jason L.¹,Petersen Charisse¹,Gogokhia Lasha¹^ORCID,Bell Rickesha¹^ORCID,Delgado Julio C.¹²^ORCID,Cox James³⁴^ORCID,Voth Warren¹^ORCID,Brown Jessica¹^ORCID,Stillman David J.¹^ORCID,O’Connell Ryan M.¹,Tebo Anne E.¹²^ORCID,Round June L.¹^ORCID

Affiliation:

1. Division of Microbiology and Immunology, Department of Pathology, University of Utah School of Medicine, Salt Lake City, UT 84112, USA.

2. ARUP Laboratories, 500 Chipeta Way, Salt Lake City, UT 84108, USA.

3. Metabolomics Core, University of Utah Health Sciences Center, Salt Lake City, UT 84112, USA.

4. Department of Biochemistry, University of Utah School of Medicine, Salt Lake City, UT 84112, USA.

Abstract

Saccharomyces cerevisiae induces purine metabolism in mouse intestinal epithelia, resulting in an exacerbation of colitis.

Funder

T32 genetics training

NIH New Innovator Award

National Heart, Lung, and Blood Institute

American Cancer Society Research

Kimmel Scholars Award

Edward Mallinckrodt Jr. Foundation

NSF CAREER

Packard Fellowships in Science and Engineering

National Institute of Allergy and Infectious Disease

NIH Innovator Award

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine

Reference57 articles.

1. An Integrative View of Microbiome-Host Interactions in Inflammatory Bowel Diseases

2. The Treatment-Naive Microbiome in New-Onset Crohn’s Disease

3. Molecular-phylogenetic characterization of microbial community imbalances in human inflammatory bowel diseases

4. A microbial symbiosis factor prevents intestinal inflammatory disease

5. Treg induction by a rationally selected mixture of Clostridia strains from the human microbiota

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