IFNAR1 signaling in NK cells promotes persistent virus infection-Reference-Cited by-同舟云学术

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IFNAR1 signaling in NK cells promotes persistent virus infection

Published:2021-03-26 Issue:13 Volume:7 Page:
ISSN:2375-2548
Container-title:Science Advances
language:en
Short-container-title:Sci. Adv.

Author:

Huang Zhe¹^ORCID,Kang Seung Goo¹²^ORCID,Li Yunqiao¹,Zak Jaroslav¹^ORCID,Shaabani Namir¹^ORCID,Deng Kaiyuan¹³,Shepherd Jovan¹^ORCID,Bhargava Raag¹,Teijaro John R.¹^ORCID,Xiao Changchun¹^ORCID

Affiliation:

1. Department of Immunology and Microbiology, The Scripps Research Institute, La Jolla, CA 92037, USA.

2. Department of Molecular Bioscience/Institute of Bioscience and Biotechnology, College of Biomedical Science, Kangwon National University, Chuncheon, Republic of Korea.

3. School of Medicine, Nankai University, Tianjin 30071, China.

Abstract

IFNAR1 signaling in NK cells compromises cellular and humoral immune responses to hamper virus control.

Funder

National Institutes of Health

National Institutes of Healthe

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference61 articles.

1. Immune activation and inflammation in HIV-1 infection: causes and consequences

2. Innate Immune Activation in Primary HIV‐1 Infection

3. HIV-Associated Immune Activation: From Bench to Bedside

4. Interleukin-10 determines viral clearance or persistence in vivo

5. Restoring function in exhausted CD8 T cells during chronic viral infection

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