T RM maintenance is regulated by tissue damage via P2RX7-Reference-Cited by-同舟云学术

T RM maintenance is regulated by tissue damage via P2RX7

Published:2018-12-14 Issue:30 Volume:3 Page:
ISSN:2470-9468
Container-title:Science Immunology
language:en
Short-container-title:Sci. Immunol.

Author:

Stark Regina¹²^ORCID,Wesselink Thomas H.¹,Behr Felix M.¹²^ORCID,Kragten Natasja A. M.¹,Arens Ramon³^ORCID,Koch-Nolte Friedrich⁴^ORCID,van Gisbergen Klaas P. J. M.¹²^ORCID,van Lier René A. W.¹²

Affiliation:

1. Department of Hematopoiesis, Sanquin Research and Landsteiner Laboratory, Amsterdam UMC, University of Amsterdam, Amsterdam, Netherlands.

2. Department of Experimental Immunology, Amsterdam UMC, Amsterdam, Netherlands.

3. Department of Immunohematology and Blood Transfusion, Leiden University Medical Center, Leiden, Netherlands.

4. Institute of Immunology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.

Abstract

The recognition of tissue damage via P2RX7 reduces the persistence of tissue-resident memory T cells (T RM ).

Funder

Alexander von Humboldt-Stiftung

Landsteiner Foundation for Blood Transfusion Research

Nederlandse Organisatie voor Wetenschappelijk Onderzoek

Publisher

American Association for the Advancement of Science (AAAS)

Subject

General Medicine,Immunology

Reference60 articles.

1. Memory T cells in nonlymphoid tissue that provide enhanced local immunity during infection with herpes simplex virus

2. Skin infection generates non-migratory memory CD8+ TRM cells providing global skin immunity

3. Long-lived epithelial immunity by tissue-resident memory T (TRM) cells in the absence of persisting local antigen presentation

4. Resident memory CD8 T cells trigger protective innate and adaptive immune responses

5. Skin-resident memory CD8 + T cells trigger a state of tissue-wide pathogen alert

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