Purinergic Control of T Cell Activation by ATP Released Through Pannexin-1 Hemichannels-Reference-Cited by-同舟云学术

Purinergic Control of T Cell Activation by ATP Released Through Pannexin-1 Hemichannels

Published:2008-09-30 Issue:39 Volume:1 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Schenk Ursula¹,Westendorf Astrid M.²,Radaelli Enrico³,Casati Anna¹⁴,Ferro Micol¹,Fumagalli Marta⁵,Verderio Claudia⁶,Buer Jan²,Scanziani Eugenio³,Grassi Fabio¹

Affiliation:

1. Institute for Research in Biomedicine, 6500 Bellinzona, Switzerland.

2. Institute of Medical Microbiology, University Hospital Essen, 45122 Essen, Germany.

3. Department of Veterinary Pathology, Hygiene and Public Health, University of Milan, 20100 Milan, Italy.

4. Department of Biology and Genetics for Medical Sciences, University of Milan, 20100 Milan, Italy.

5. Department of Pharmacological Sciences, University of Milan, 20100 Milan, Italy.

6. Department of Medical Pharmacology, CNR Institute of Neuroscience, University of Milan, 20100 Milan, Italy.

Abstract

Pannexin hemichannel–mediated release of ATP provides an autocrine, costimulatory signal for T cell activation.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference72 articles.

1. Calcium Signaling Mechanisms in T Lymphocytes

2. A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function

3. Orai1 is an essential pore subunit of the CRAC channel

4. CRACM1 Is a Plasma Membrane Protein Essential for Store-Operated Ca 2+ Entry

5. Molecular identification of the CRAC channel by altered ion selectivity in a mutant of Orai

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