Histone deacetylase 3 supports endochondral bone formation by controlling cytokine signaling and matrix remodeling-Reference-Cited by-同舟云学术

Histone deacetylase 3 supports endochondral bone formation by controlling cytokine signaling and matrix remodeling

Published:2016-08-09 Issue:440 Volume:9 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

Carpio Lomeli R.¹²,Bradley Elizabeth W.³,McGee-Lawrence Meghan E.⁴⁵,Weivoda Megan M.⁶,Poston Daniel D.²⁷,Dudakovic Amel³,Xu Ming⁸,Tchkonia Tamar⁸,Kirkland James L.⁸,van Wijnen Andre J.²³,Oursler Merry Jo²⁶,Westendorf Jennifer J.²³

Affiliation:

1. Mayo Graduate School, Mayo Clinic, Rochester, MN 55905, USA.

2. Department of Biochemistry and Molecular Biology, Mayo Clinic, Rochester, MN 55905, USA.

3. Department of Orthopedic Surgery, Mayo Clinic, Rochester, MN 55905, USA.

4. Department of Cellular Biology and Anatomy, Medical College of Georgia, Augusta University, Augusta, GA 30912, USA.

5. Institute of Regenerative and Reparative Medicine, Augusta University, Augusta, GA 30912, USA.

6. Division of Endocrinology, Diabetes, Metabolism and Nutrition, Department of Medicine, Mayo Clinic, Rochester, MN 55905, USA.

7. Creighton University, Omaha, NE 68102, USA.

8. Robert and Arlene Kogod Center on Aging, Mayo Clinic, Rochester, MN 55905, USA.

Abstract

Histone deacetylase 3 prevents chondrocytes from producing inflammatory mediators that prevent bone development.

Funder

NIH

Ted Nash Foundation

Connor Group

charitable foundation of William and Karen Eby

Mayo Clinic

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference92 articles.

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3. RECENT RESEARCH ON THE GROWTH PLATE: Impact of inflammatory cytokines on longitudinal bone growth

4. The restricted potential for recovery of growth plate chondrogenesis and longitudinal bone growth following exposure to pro-inflammatory cytokines

5. The pathophysiology of the growth plate in juvenile idiopathic arthritis

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