Stabilization of VEGFR2 Signaling by Cerebral Cavernous Malformation 3 Is Critical for Vascular Development-Reference-Cited by-同舟云学术

Stabilization of VEGFR2 Signaling by Cerebral Cavernous Malformation 3 Is Critical for Vascular Development

Published:2010-04-06 Issue:116 Volume:3 Page:
ISSN:1945-0877
Container-title:Science Signaling
language:en
Short-container-title:Sci. Signal.

Author:

He Yun¹,Zhang Haifeng¹,Yu Luyang¹,Gunel Murat¹²,Boggon Titus J.³,Chen Hong¹⁴,Min Wang¹

Affiliation:

1. Interdepartmental Program in Vascular Biology and Therapeutics, Department of Pathology, Yale University School of Medicine, 10 Amistad Street, New Haven, CT 06520, USA.

2. Department of Neurosurgery, Yale University School of Medicine, New Haven, CT 06520, USA.

3. Department of Pharmacology, Yale University School of Medicine, New Haven, CT 06520, USA.

4. Cardiovascular Biology Research Program, Oklahoma Medical Research Foundation, Oklahoma City, OK 73104, USA.

Abstract

CCM3, which is associated with inherited cerebrovascular defects, prevents ligand-mediated degradation of a receptor involved in blood vessel formation.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Cell Biology,Molecular Biology,Biochemistry

Reference64 articles.

1. 131 cases of cavernous angioma (cavernomas) of the CNS, discovered by retrospective analysis of 24,535 autopsies.;Otten P.;Neurochirurgie,1989

2. Cerebral Cavernous Malformations

3. Cerebral cavernous malformation: new molecular and clinical insights

4. Mutations in the Gene Encoding KRIT1, a Krev-1/rap1a Binding Protein, Cause Cerebral Cavernous Malformations (CCM1)

5. KRIT1 is mutated in hyperkeratotic cutaneous capillary-venous malformation associated with cerebral capillary malformation

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