Comment on “Aberrant type 1 immunity drives susceptibility to mucosal fungal infections”-Reference-Cited by-同舟云学术

Comment on “Aberrant type 1 immunity drives susceptibility to mucosal fungal infections”

Published:2021-09-17 Issue:6561 Volume:373 Page:
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Puel Anne¹²³⁴^ORCID,Casanova Jean-Laurent¹²³⁴^ORCID

Affiliation:

1. Laboratory of Human Genetics of Infectious Diseases, Necker Branch, INSERM, Necker Hospital for Sick Children, Paris, France.

2. University of Paris, Imagine Institute, Paris, France.

3. St. Giles Laboratory of Human Genetics of Infectious Diseases, Rockefeller Branch, The Rockefeller University, New York, NY, USA.

4. Howard Hughes Medical Institute, The Rockefeller University, New York, NY, USA.

Abstract

Break et al . (Research Articles, 15 January 2021, eaay5731) suggest that chronic mucocutaneous candidiasis in humans with inborn errors of AIRE and autoimmune polyendocrine syndrome type 1 is due to excessive interferon-γ production and not to autoantibodies neutralizing interleukin-17 cytokines. We argue that both claims are not conclusively supported by their data and are at odds with 35 years of study.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference15 articles.

1. Autoimmune Polyendocrine Syndromes

2. Projection of an Immunological Self Shadow Within the Thymus by the Aire Protein

3. Aberrant type 1 immunity drives susceptibility to mucosal fungal infections

4. Autoantibodies against type I IFNs in patients with life-threatening COVID-19

5. Chronic Mucocutaneous Candidiasis in Humans with Inborn Errors of Interleukin-17 Immunity

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