Abnormal Behavior Associated with a Point Mutation in the Structural Gene for Monoamine Oxidase A-Reference-Cited by-同舟云学术

Abnormal Behavior Associated with a Point Mutation in the Structural Gene for Monoamine Oxidase A

Published:1993-10-22 Issue:5133 Volume:262 Page:578-580
ISSN:0036-8075
Container-title:Science
language:en
Short-container-title:Science

Author:

Brunner H. G.¹,Nelen M.²,Breakefield X. O.³,Ropers H. H.²,van Oost B. A.²

Affiliation:

1. Department of Human Genetics, University Hospital Nijmegen, Geert Grooteplein 20, 6525 GA Nijmegen, the Netherlands.

2. Department of Human Genetics, University Hospital Nijmegen, 6500 HB Nijmegen, the Netherlands.

3. Neuroscience Center, Massachusetts General Hospital, Charlestown, MA 02129.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference43 articles.

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2. BERGER, W, ISOLATION OF A CANDIDATE GENE FOR NORRIE DISEASE BY POSITIONAL CLONING, NATURE GENETICS 1: 199 (1992).

3. BLEEKERWAGEMAKE.EM, NORRIE DISEASE AS PART OF A COMPLEX SYNDROME EXPLAINED BY A SUBMICROSCOPIC DELETION OF THE X-CHROMOSOME, OPHTHALMIC PAEDIATRICS AND GENETICS 9: 137 (1988).

4. BLIER, P, MODIFICATION OF SEROTONERGIC AND NORADRENERGIC NEUROTRANSMISSIONS BY REPEATED ADMINISTRATION OF MONOAMINE-OXIDASE INHIBITORS - ELECTROPHYSIOLOGICAL STUDIES IN THE RAT CENTRAL-NERVOUS-SYSTEM, JOURNAL OF PHARMACOLOGY AND EXPERIMENTAL THERAPEUTICS 237: 987 (1986).

5. A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

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