Restoration by Calmodulin of a Ca 2+ -Dependent K + Current Missing in a Mutant of Paramecium
Author:
Affiliation:
1. Laboratory of Molecular Biology and Department of Genetics, University of Wisconsin, Madison, WI 53706.
2. Department of Biochemistry, University of Wisconsin, Madison, WI 53706.
Publisher
American Association for the Advancement of Science (AAAS)
Subject
Multidisciplinary
Reference20 articles.
1. BLAIR, LAC, DEVELOPMENTAL ACQUISITION OF CA-2+-SENSITIVITY BY K+ CHANNELS IN SPINAL NEURONS, NATURE 315: 329 (1985).
2. CHEN, P.S., MICRODETERMINATION OF PHOSPHORUS, ANALYTICAL CHEMISTRY 28: 1756 (1956).
3. DEPEYER, J.E., CA-2+-ACTIVATED K+ CONDUCTANCE IN INTERNALLY PERFUSED SNAIL NEURONS IS ENHANCED BY PROTEIN-PHOSPHORYLATION, PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA-BIOLOGICAL SCIENCES 79: 4207 (1982).
4. EWALD, D.A., MODULATION OF SINGLE CA-2+-DEPENDENT K+-CHANNEL ACTIVITY BY PROTEIN-PHOSPHORYLATION, NATURE 315: 503 (1985).
5. GUERINI, D, INFLUENCE OF TEMPERATURE AND DENATURING AGENTS ON THE STRUCTURAL STABILITY OF CALMODULIN - A-H-1-NUCLEAR MAGNETIC-RESONANCE STUDY, FEBS LETTERS 164: 105 (1983).
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