Defining Protective Responses to Pathogens: Cytokine Profiles in Leprosy Lesions

Author:

Yamamura Masahiro1,Uyemura Koichi1,Deans Robert J.2,Weinberg Kenneth3,Rea Thomas H.4,Bloom Barry R.5,Modlin Robert L6

Affiliation:

1. Division of Dermatology, UCLA School of Medicine, Los Angeles, CA 90024.

2. Department of Neurology, University of Southern California School of Medicine, Los Angeles, CA 90033.

3. Childrens Hospital Los Angeles, Los Angeles, CA 90054, and Department of Pediatrics, University of Southern California School of Medicine, Los Angeles, CA 90033.

4. Section of Dermatology, University of Southern California School of Medicine, Los Angeles, CA 90033.

5. Howard Hughes Medical Institute, Albert Einstein College of Medicine, Bronx, NY 10461.

6. Division of Dermatology, and Department of Microbiology and Immunology, UCLA School of Medicine, Los Angeles, CA 90033.

Publisher

American Association for the Advancement of Science (AAAS)

Subject

Multidisciplinary

Reference17 articles.

1. ARNOLDI, J, IMMUNOHISTOLOGIC ASSESSMENT OF CYTOKINE PRODUCTION OF INFILTRATING CELLS IN VARIOUS FORMS OF LEPROSY, AMERICAN JOURNAL OF PATHOLOGY 137: 749 (1990).

2. Vaccines for the Third World

3. Analysis of naturally occurring delayed-type hypersensitivity reactions in leprosy by in situ hybridization.

4. Reciprocal expression of interferon gamma or interleukin 4 during the resolution or progression of murine leishmaniasis. Evidence for expansion of distinct helper T cell subsets.

5. MODLIN, R.L., INSITU CHARACTERIZATION OF LYMPHOCYTE-T SUBSETS IN THE REACTIONAL STATES OF LEPROSY, CLINICAL AND EXPERIMENTAL IMMUNOLOGY 53: 17 (1983).

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