Proinflammatory Response in Human Keratinocytes Stimulated with Ligands for TLR2, TLR1, and TLR6-Reference-Cited by-同舟云学术

Proinflammatory Response in Human Keratinocytes Stimulated with Ligands for TLR2, TLR1, and TLR6

Published:2010 Issue:1 Volume:56 Page:50-55
ISSN:0022-6769
Container-title:Juntendo Medical Journal
language:en
Short-container-title:

Author:

TUAN VU ANH¹²,TAKAI TOSHIRO¹,ANH LE TUAN¹²,KINOSHITA HIROKAZU¹²,KAMIJO SEIJI¹,HARA MUTSUKO¹,WANG XIAO-LING¹,OGAWA TAKASUKE¹²,SUTO HAJIME¹²,IKEDA SHIGAKU¹²,OGAWA HIDEOKI¹,OKUMURA KO¹

Affiliation:

1. Allergy (Atopy) Research Center, Juntedo University Graduate School of Medicine

2. Department of Dermatology and Allergology, Juntedo University Graduate School of Medicine

Publisher

The Jutendo Medical Journal

Link

https://www.jstage.jst.go.jp/article/pjmj/56/1/56_50/_pdf

Reference30 articles.

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2. 2) PalmerCN, IrvineAD, Terron-KwiatkowskiA, et al: Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis. Nat Genet, 2006;38: 441-446.

3. 3) WalleyAJ, ChavanasS, MoffattMF, et al: Gene polymorphism in Netherton and common atopic disease. Nat Genet, 2001;29: 175-178.

4. 4) VasilopoulosY, CorkMJ, TeareD, et al: A nonsynonymous substitution of cystatin A, a cysteine protease inhibitor of house dust mite protease, leads to decreased mRNA stability and shows a significant association with atopic dermatitis. Allergy, 2007;62: 514-519.

5. 5) AlyR, MaibachHI, ShinefieldHR: Microbial flora of atopic dermatitis. Arch Dermatol, 1977;113: 780-782.

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