Synergistic Signaling of TLR and IFNα/β Facilitates Escape of IL-18 Expression from Endotoxin Tolerance-Reference-Cited by-同舟云学术

Synergistic Signaling of TLR and IFNα/β Facilitates Escape of IL-18 Expression from Endotoxin Tolerance

Published:2020-03-01 Issue:5 Volume:201 Page:526-539
ISSN:1073-449X
Container-title:American Journal of Respiratory and Critical Care Medicine
language:en
Short-container-title:Am J Respir Crit Care Med

Author:

Verweyen Emely¹,Holzinger Dirk²,Weinhage Toni¹,Hinze Claas¹^ORCID,Wittkowski Helmut¹,Pickkers Peter³^ORCID,Albeituni Sabrin⁴,Verbist Katherine⁴,Nichols Kim E.⁴^ORCID,Schulert Grant⁵⁶^ORCID,Grom Alexei⁵⁶,Foell Dirk¹,Kessel Christoph¹^ORCID

Affiliation:

1. Department of Pediatric Rheumatology and Immunology, University Children’s Hospital, Münster, Germany

2. Department of Pediatric Hematology–Oncology, University of Duisburg–Essen, Essen, Germany

3. Department of Intensive Care Medicine, Radboud University Medical Center, Nijmegen, the Netherlands

4. Division of Cancer Predisposition, St. Jude Children’s Research Hospital, Memphis, Tennessee; and

5. Department of Rheumatology, Cincinnati Children’s Hospital Medical Center and

6. Department of Pediatrics, University of Cincinnati College of Medicine, Cincinnati, Ohio

Publisher

American Thoracic Society

Subject

Critical Care and Intensive Care Medicine,Pulmonary and Respiratory Medicine

Link

https://www.atsjournals.org/doi/pdf/10.1164/rccm.201903-0659OC

Reference69 articles.

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2. Balance between Hyperinflammation and Immunosuppression in Sepsis

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4. Precision Immunotherapy for Sepsis

5. Interleukin-18 diagnostically distinguishes and pathogenically promotes human and murine macrophage activation syndrome

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