A biochemical mechanism for the oncogenic potential of the p110 catalytic subunit of phosphoinositide 3-kinase
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference32 articles.
1. The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism
2. PI3K pathway alterations in cancer: variations on a theme
3. Class IA phosphoinositide 3-kinases are obligate p85-p110 heterodimers
4. Heterodimeric Phosphoinositide 3-Kinase Consisting of p85 and p110β Is Synergistically Activated by the βγ Subunits of G Proteins and Phosphotyrosyl Peptide
5. The p110 isoform of phosphoinositide 3-kinase signals downstream of G protein-coupled receptors and is functionally redundant with p110
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