Consequences of NMO-IgG binding to aquaporin-4 in neuromyelitis optica
Author:
Publisher
Proceedings of the National Academy of Sciences
Subject
Multidisciplinary
Reference5 articles.
1. Molecular outcomes of neuromyelitis optica (NMO)-IgG binding to aquaporin-4 in astrocytes
2. Aquaporin-4–binding autoantibodies in patients with neuromyelitis optica impair glutamate transport by down-regulating EAAT2
3. Evidence against Cellular Internalization in Vivo of NMO-IgG, Aquaporin-4, and Excitatory Amino Acid Transporter 2 in Neuromyelitis Optica
4. Complement-dependent Cytotoxicity in Neuromyelitis Optica Requires Aquaporin-4 Protein Assembly in Orthogonal Arrays
5. Aquaporin-4 orthogonal arrays of particles are the target for neuromyelitis optica autoantibodies
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1. From Homeostasis to Pathology: Decoding the Multifaceted Impact of Aquaporins in the Central Nervous System;International Journal of Molecular Sciences;2023-09-20
2. Rituximab abrogates aquaporin-4–specific germinal center activity in patients with neuromyelitis optica spectrum disorders;Proceedings of the National Academy of Sciences;2022-06-06
3. Autopsy Case of Meningoencephalomyelitis Associated With Glial Fibrillary Acidic Protein Antibody;Neurology - Neuroimmunology Neuroinflammation;2021-10-12
4. Joint Application of Magnetic Resonance Imaging and Biochemical Biomarkers in Diagnosis of Multiple Sclerosis;Current Medicinal Chemistry;2020-11-24
5. Functional Role of Aquaporins in the Nervous System under Normal and Pathological Conditions;Neurochemical Journal;2020-01
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