Transient cardiac expression of constitutively active G q leads to hypertrophy and dilated cardiomyopathy by calcineurin-dependent and independent pathways-Reference-Cited by-同舟云学术

Transient cardiac expression of constitutively active G q leads to hypertrophy and dilated cardiomyopathy by calcineurin-dependent and independent pathways

Published:1998-11-10 Issue:23 Volume:95 Page:13893-13898
ISSN:0027-8424
Container-title:Proceedings of the National Academy of Sciences
language:en
Short-container-title:Proceedings of the National Academy of Sciences

Author:

Mende U.,Kagen A.,Cohen A.,Aramburu J.,Schoen F. J.,Neer E. J.

Publisher

Proceedings of the National Academy of Sciences

Subject

Multidisciplinary

Reference25 articles.

1. The role of protein kinases in adaptational growth of the heart

2. Myocardial expression of a constitutively active alpha 1B-adrenergic receptor in transgenic mice induces cardiac hypertrophy.

3. Transgenic Mice with Cardiac Overexpression of α1B-Adrenergic Receptors

4. Overexpression of angiotensin AT1 receptor transgene in the mouse myocardium produces a lethal phenotype associated with myocyte hyperplasia and heart block

5. Targeting the Receptor-G q Interface to Inhibit in Vivo Pressure Overload Myocardial Hypertrophy

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