Attenuation of nuclear factor kappa B (NF-kappaB) promotes apoptosis of kidney epithelial cells: a potential mechanism of mercury-induced nephrotoxicity.-Reference-Cited by-同舟云学术

Attenuation of nuclear factor kappa B (NF-kappaB) promotes apoptosis of kidney epithelial cells: a potential mechanism of mercury-induced nephrotoxicity.

Published:2002-10 Issue:suppl 5 Volume:110 Page:819-822
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environmental Health Perspectives

Author:

Woods James S¹,Dieguez-Acuña Francisco J¹,Ellis Maureen E¹,Kushleika John¹,Simmonds P Lynne¹

Affiliation:

1. Center for Ecogenetics and Environmental Health, Department of Environmental Health, University of Washington, 4225 Roosevelt Way, Suite 100, Seattle, WA 98105, USA. jwoods@u.washington.edu

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference21 articles.

1. New insights into the role of nuclear factor-κB, a ubiquitous transcription factor, in the initiation of disease;Chen F;Clin Chem,1999

2. N-terminal DNA-binding domains contribute to differential DNA-binding specificities of NF-kappa B p50 and p65.

3. Activation of NF-κB in Normal Rat Kidney Epithelial (NRK52E) Cells Is Mediated via a Redox-Insensitive, Calcium-Dependent Pathway

4. Mercuric Ion Attenuates Nuclear Factor-κB Activation and DNA Binding in Normal Rat Kidney Epithelial Cells: Implications for Mercury-Induced Nephrotoxicity

5. Inhibition of NF-κB–DNA binding by mercuric ion: utility of the non-thiol reductant, tris(2-carboxyethyl)phosphine hydrochloride (TCEP), on detection of impaired NF-κB–DNA binding by thiol-directed agents

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