Assessing the Association of Mitochondrial Function and Inflammasome Activation in Murine Macrophages Exposed to Select Mitotoxic Tri-Organotin Compounds-Reference-Cited by-同舟云学术

Assessing the Association of Mitochondrial Function and Inflammasome Activation in Murine Macrophages Exposed to Select Mitotoxic Tri-Organotin Compounds

Published:2021-04 Issue:4 Volume:129 Page:047015
ISSN:0091-6765
Container-title:Environmental Health Perspectives
language:en
Short-container-title:Environ Health Perspect

Author:

Childers Gabrielle M.¹,Perry Caroline A.¹,Blachut Barbara¹,Martin Negin²,Bortner Carl D.³,Sieber Stella⁴,Li Jian-Liang⁵,Fessler Michael B.⁶,Harry G. Jean¹

Affiliation:

1. Molecular Toxicology Branch, National Institute of Environmental Health Sciences (NIEHS), National Institutes of Health (NIH), Department of Health and Human Services (DHHS), Research Triangle Park, North Carolina, USA

2. Laboratory of Neurobiology, NIEHS, NIH, DHHS, Research Triangle Park, North Carolina, USA

3. Signal Transduction Laboratory, NIEHS, NIH, DHHS, Research Triangle Park, North Carolina, USA

4. Molecular Genomics Core Laboratory, NIEHS, NIH, DHHS, Research Triangle Park, North Carolina, USA

5. Integrative Bioinformatics Support Group, NIEHS, NIH, DHHS, Research Triangle Park, North Carolina, USA

6. Immunity, Inflammation, and Disease Laboratory, NIEHS, NIH, DHHS, Research Triangle Park, North Carolina, USA

Publisher

Environmental Health Perspectives

Subject

Health, Toxicology and Mutagenesis,Public Health, Environmental and Occupational Health

Reference161 articles.

1. Redox Regulation of NLRP3 Inflammasomes: ROS as Trigger or Effector?

2. The action of 5-coordinate triorganotin compounds on rat liver mitochondria

3. Oxidative phosphorylation. Halide-dependent and halide-independent effects of triorganotin and trioganolead compounds on mitochondrial functions

4. Cellular and molecular effects of trimethyltin and triethyltin: Relevance to organotin neurotoxicity

5. Interactions of trimethyl tin (TMT) with rat primary astrocyte cultures: altered uptake and efflux of rubidium,l-glutamate andD-aspartate

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