Testing the role of apoA-I, HDL, and cholesterol efflux in the atheroprotective action of low-level apoE expression

Author:

Thorngate Fayanne E.,Yancey Patricia G.,Kellner-Weibel Ginny,Rudel Lawrence L.,Rothblat George H.,Williams David L.

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Biochemistry

Reference46 articles.

1. The response-to-retention hypothesis of early atherogenesis;Williams;Arterioscler. Thromb. Vasc. Biol.,1995

2. Low levels of extrahepatic non-macrophage apoE inhibit atherosclerosis without correcting hypercholesterolemia in apoE-deficient mice;Thorngate;Arterioscler. Thromb. Vasc. Biol.,2000

3. Low-dose expression of a human apolipoprotein E transgene in macrophages restores cholesterol efflux capacity of apolipoprotein E-deficient mouse plasma;Zhu;Proc. Natl. Acad. Sci. USA.,1998

4. Retroviral gene therapy in ApoE-deficient mice Apo E expression in the artery wall reduces early foam cell lesion formation;Hasty;Basic Sci. Reports.,1999

5. Importance of different pathways of cellular cholesterol efflux;Yancey;Arterioscler. Thromb. Vasc. Biol.,2003

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