The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis-Reference-Cited by-同舟云学术

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The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis

Published:2015-09-14 Issue:9 Volume:11 Page:e1005155
ISSN:1553-7374
Container-title:PLOS Pathogens
language:en
Short-container-title:PLoS Pathog

Author:

Guo Sheng,Yang Chengying,Diao Bo,Huang Xiaoyong,Jin Meihua,Chen Lili,Yan Weiming,Ning Qin,Zheng Lixin,Wu Yuzhang,Chen Yongwen

Publisher

Public Library of Science (PLoS)

Subject

Virology,Genetics,Molecular Biology,Immunology,Microbiology,Parasitology

Reference49 articles.

1. Acute-on-chronic liver failure: consensus recommendations of the Asian Pacific Association for the study of the liver (APASL);SK Sarin;Hepatol Int,2009

2. Fulminant viral hepatitis: molecular and cellular basis, and clinical implications;M Liu;Expert Rev Mol Med,2001

3. The Fgl2/fibroleukin prothrombinase contributes to immunologically mediated thrombosis in experimental and human viral hepatitis;PA Marsden;J Clin Invest,2003

4. C5aR, TNF-α and Fgl2 contribute to coagulation and complement activation in virus induced fulminant hepatitis;J Liu;J Hepatol,2015

5. C5a/C5aR pathway is essential for the pathogenesis of murine viral fulminant hepatitis by way of potentiating Fgl2/fibroleukin expression;GL Xu;Hepatology,2014

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