Arginase inhibition by rhaponticin increases L-arginine concentration that contributes to Ca2+-dependent eNOS activation
Author:
Affiliation:
1. Department of Biological Sciences, Kangwon National University, Chuncheon 24341, Korea
2. Department of Anesthesiology and Pain Medicine, Yonsei University Wonju College of Medicine, Wonju 26426, Korea
Funder
Ministry of Education, Science and Technology
Publisher
Korean Society for Biochemistry and Molecular Biology - BMB Reports
Subject
Molecular Biology,Biochemistry,General Medicine
Link
http://pdf.medrang.co.kr/BMB/2021/054/BMB054-10-04.pdf
Reference21 articles.
1. Role of the arginine-nitric oxide pathway in the regulation of vascular smooth muscle cell proliferation
2. Regulatory role of arginase I and II in nitric oxide, polyamine, and proline syntheses in endothelial cells
3. Arginase Reciprocally Regulates Nitric Oxide Synthase Activity and Contributes to Endothelial Dysfunction in Aging Blood Vessels
4. Ischemia‐reperfusion selectively impairs nitric oxide‐ mediated dilation in coronary arterioles: counteracting role of arginase
5. Arginase inhibition mediates cardioprotection during ischaemia-reperfusion
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