Anisomycin protects against sepsis by attenuating IκB kinase-dependent NF-κB activation and inflammatory gene expression-Reference-Cited by-同舟云学术

Anisomycin protects against sepsis by attenuating IκB kinase-dependent NF-κB activation and inflammatory gene expression

Published:2021-11-30 Issue:11 Volume:54 Page:545-550
ISSN:1976-670X
Container-title:BMB Reports
language:en
Short-container-title:BMB Rep

Author:

Park Gyoung Lim¹,Park Minkyung¹,Min Jeong-Ki²,Park Young-Jun¹,Chung Su Wol³,Lee Seon-Jin¹^ORCID

Affiliation:

1. Environmental Disease Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Daejeon 34141, Korea

2. Biotherapeutics Translational Research Center, Korea Research Institute of Bioscience and Biotechnology (KRIBB), Daejeon 34141, Korea

3. School of Biological Sciences, University of Ulsan, Ulsan 44610, 4Department of Functional Genomics, University of Science and Technology (UST), Daejeon 34113, Korea

Funder

National Research Foundation of Korea

the KIST Institutional Program

the KRIBB Research Initiative Program

Publisher

Korean Society for Biochemistry and Molecular Biology - BMB Reports

Subject

Molecular Biology,Biochemistry,General Medicine

Link

http://pdf.medrang.co.kr/BMB/2021/054/BMB054-11-02.pdf

Reference16 articles.

1. Sepsis and septic shock

2. The Pathogenesis of Sepsis and Potential Therapeutic Targets

3. In vivo toxicological evaluation of Anisomycin

4. Carbon Monoxide Confers Protection in Sepsis by Enhancing Beclin 1-Dependent Autophagy and Phagocytosis

5. TXNIP Deficiency Exacerbates Endotoxic Shock via the Induction of Excessive Nitric Oxide Synthesis

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